ACAD-11 inhibitors constitute a diverse group of chemicals targeting various aspects of fatty acid metabolism and cellular energetics. Etomoxir and Perhexiline are inhibitors of carnitine palmitoyltransferase 1 (CPT-1), disrupting fatty acid oxidation and indirectly influencing ACAD-11 by modulating substrate availability for its reactions. Ranolazine, Trimetazidine, and Mildronate are fatty acid oxidation inhibitors, altering cellular energetics and substrate availability for ACAD-11 reactions. Etofibrate and Fenofibrate, as peroxisome proliferator-activated receptor alpha (PPAR-α) agonists, modulate lipid metabolism, indirectly affecting ACAD-11 by altering the expression of genes related to fatty acid metabolism and substrate availability. Bupivacaine and Diltiazem, as ion channel modulators, impact cellular excitability, influencing ACAD-11 indirectly by altering cellular conditions and energetics.
CB-839, a glutaminase inhibitor, disrupts glutamine metabolism, indirectly influencing ACAD-11 by altering the availability of metabolites that cross-talk with fatty acid metabolism. This diverse array of inhibitors provides valuable tools for investigating the intricate regulatory networks involving ACAD-11, offering insights into its role in fatty acid metabolism and cellular energetics.
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| Product Name | CAS # | Catalog # | QUANTITY | Price | Citations | RATING |
|---|---|---|---|---|---|---|
(+)-Etomoxir sodium salt | 828934-41-4 | sc-215009 sc-215009A | 5 mg 25 mg | $151.00 $506.00 | 3 | |
Etomoxir, a carnitine palmitoyltransferase 1 (CPT-1) inhibitor, disrupts fatty acid oxidation. Its impact on CPT-1-mediated processes indirectly influences ACAD-11 by altering the availability of substrate for ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
rac Perhexiline Maleate | 6724-53-4 | sc-460183 | 10 mg | $188.00 | ||
Perhexiline, a carnitine palmitoyltransferase 1 (CPT-1) inhibitor, disrupts fatty acid oxidation. Its influence on CPT-1-mediated processes indirectly affects ACAD-11 by modulating the availability of substrate for ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
Ranolazine | 95635-55-5 | sc-212769 | 1 g | $109.00 | 3 | |
Ranolazine, a fatty acid oxidation inhibitor, modulates ion channel activity. Its impact on fatty acid oxidation indirectly influences ACAD-11 by altering cellular energetics and the availability of substrates for ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
Trimetazidine Dihydrochloride | 13171-25-0 | sc-220334 | 10 mg | $213.00 | ||
Trimetazidine, a fatty acid oxidation inhibitor, modulates cellular energetics. Its influence on fatty acid oxidation indirectly affects ACAD-11 by altering substrate availability and cellular conditions that impact ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
Emetine | 483-18-1 | sc-470668 sc-470668A sc-470668B sc-470668C | 1 mg 10 mg 50 mg 100 mg | $440.00 $900.00 $1400.00 $2502.00 | ||
Etofibrate, a peroxisome proliferator-activated receptor alpha (PPAR-α) agonist, influences lipid metabolism. Its impact on PPAR-α-mediated pathways indirectly influences ACAD-11 by altering the expression of genes related to fatty acid metabolism and substrate availability for ACAD-11-mediated reactions. | ||||||
Diltiazem | 42399-41-7 | sc-204726 sc-204726A | 1 g 5 g | $209.00 $464.00 | 4 | |
Diltiazem, a calcium channel blocker, modulates cellular excitability. Its influence on calcium channel activity indirectly affects ACAD-11 by altering cellular conditions and energetics, impacting the availability of substrates for ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
Meldonium | 76144-81-5 | sc-207887 | 100 mg | $455.00 | 1 | |
Mildronate, a fatty acid oxidation modulator, influences cellular energetics. Its impact on fatty acid metabolism indirectly influences ACAD-11 by altering substrate availability and cellular conditions that impact ACAD-11-mediated reactions in fatty acid metabolism. | ||||||
Fenofibrate | 49562-28-9 | sc-204751 | 5 g | $41.00 | 9 | |
Fenofibrate, a peroxisome proliferator-activated receptor alpha (PPAR-α) agonist, influences lipid metabolism. Its influence on PPAR-α-mediated pathways indirectly influences ACAD-11 by altering the expression of genes related to fatty acid metabolism and substrate availability for ACAD-11-mediated reactions. | ||||||
CB 839 | 1439399-58-2 | sc-507354 | 10 mg | $140.00 | ||
CB-839, a glutaminase inhibitor, disrupts glutamine metabolism. Its impact on glutaminase-mediated processes indirectly influences ACAD-11 by altering the availability of metabolites that cross-talk with fatty acid metabolism, impacting ACAD-11-mediated reactions. | ||||||