MLC1 inhibitors target the MLC1 protein, which is associated with cellular processes like differentiation, proliferation, and apoptosis. MLC1 is known to be involved in leukemogenesis, suggesting its potential role in cancer-related pathways. Inhibitors targeting MLC1 would aim to modulate these processes, possibly by disrupting MLC1's interactions with other proteins or its ability to regulate gene expression. One approach to inhibit MLC1 could involve small molecules that interfere with its interaction domains, preventing it from forming complexes necessary for its function. Another strategy might be to target the regulatory mechanisms that control MLC1 expression, such as transcription factors or signaling pathways that upregulate or downregulate MLC1.
The potential inhibitors listed above, such as Rapamycin, Curcumin, and Resveratrol, demonstrate an indirect approach to modulating MLC1 activity. These compounds are known to influence signaling pathways and gene expression patterns that could, in turn, impact the function of MLC1. For example, by affecting pathways involved in cell growth and proliferation, Rapamycin might indirectly influence MLC1's role in these processes. Currently, direct inhibitors of MLC1 are not established, and the focus is on understanding its biological role and finding ways to modulate its activity indirectly. This involves a broader understanding of cellular signaling pathways and the interplay between different molecular processes. The compounds listed demonstrate the potential for chemical interventions in regulating protein functions like MLC1, highlighting the complex and interconnected nature of biological systems.
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| Product Name | CAS # | Catalog # | QUANTITY | Price | Citations | RATING |
|---|---|---|---|---|---|---|
Rapamycin | 53123-88-9 | sc-3504 sc-3504A sc-3504B | 1 mg 5 mg 25 mg | $62.00 $155.00 $320.00 | 233 | |
Rapamycin, an mTOR inhibitor, may indirectly affect MLC1 through its role in signaling pathways. | ||||||
Curcumin | 458-37-7 | sc-200509 sc-200509A sc-200509B sc-200509C sc-200509D sc-200509F sc-200509E | 1 g 5 g 25 g 100 g 250 g 1 kg 2.5 kg | $36.00 $68.00 $107.00 $214.00 $234.00 $862.00 $1968.00 | 47 | |
Curcumin can modulate various signaling pathways and gene expression, possibly influencing MLC1. | ||||||
Resveratrol | 501-36-0 | sc-200808 sc-200808A sc-200808B | 100 mg 500 mg 5 g | $60.00 $185.00 $365.00 | 64 | |
Resveratrol impacts transcription factors and signaling pathways, potentially affecting MLC1. | ||||||
D,L-Sulforaphane | 4478-93-7 | sc-207495A sc-207495B sc-207495C sc-207495 sc-207495E sc-207495D | 5 mg 10 mg 25 mg 1 g 10 g 250 mg | $150.00 $286.00 $479.00 $1299.00 $8299.00 $915.00 | 22 | |
DL-Sulforaphane might alter gene expression in a way that impacts MLC1 activity. | ||||||
Quercetin | 117-39-5 | sc-206089 sc-206089A sc-206089E sc-206089C sc-206089D sc-206089B | 100 mg 500 mg 100 g 250 g 1 kg 25 g | $11.00 $17.00 $108.00 $245.00 $918.00 $49.00 | 33 | |
Quercetin may modulate transcriptional activity and signaling pathways, potentially influencing MLC1. | ||||||
Metformin | 657-24-9 | sc-507370 | 10 mg | $77.00 | 2 | |
Metformin influences metabolic pathways, which could indirectly affect MLC1. | ||||||
Pioglitazone | 111025-46-8 | sc-202289 sc-202289A | 1 mg 5 mg | $54.00 $123.00 | 13 | |
As a PPAR gamma agonist, it may indirectly influence MLC1 activity. | ||||||
Retinoic Acid, all trans | 302-79-4 | sc-200898 sc-200898A sc-200898B sc-200898C | 500 mg 5 g 10 g 100 g | $65.00 $319.00 $575.00 $998.00 | 28 | |
Retinoic Acid, all trans affects gene expression and could indirectly influence MLC1. | ||||||
Cholecalciferol | 67-97-0 | sc-205630 sc-205630A sc-205630B | 1 g 5 g 10 g | $70.00 $160.00 $290.00 | 2 | |
Cholecalciferol regulates gene expression, potentially affecting MLC1. | ||||||
Genistein | 446-72-0 | sc-3515 sc-3515A sc-3515B sc-3515C sc-3515D sc-3515E sc-3515F | 100 mg 500 mg 1 g 5 g 10 g 25 g 100 g | $26.00 $92.00 $120.00 $310.00 $500.00 $908.00 $1821.00 | 46 | |
Genistein could affect MLC1 by influencing gene transcription processes. | ||||||