ATF-7IP inhibitors belong to a distinct chemical class that plays a pivotal role in modulating cellular processes by targeting ATF-7IP, a crucial transcription factor associated with gene regulation. ATF-7IP, also known as activating transcription factor 7-interacting protein, is involved in chromatin remodeling and transcriptional control, influencing the expression of genes implicated in diverse cellular functions. The inhibitors designed to interact with ATF-7IP act as specific molecular entities capable of binding to its active sites, thereby altering its activity and subsequent downstream effects on gene expression.
ATF-7IP inhibitors is meticulously engineered to ensure high selectivity and affinity for ATF-7IP, minimizing off-target effects. Typically, these compounds possess specific functional groups that facilitate interaction with key residues in the ATF-7IP binding pocket. The development of ATF-7IP inhibitors has been driven by a profound understanding of the structural and biochemical characteristics of ATF-7IP, enabling rational drug design to achieve optimal binding and modulation. As researchers delve into the intricacies of ATF-7IP inhibition, advancements in medicinal chemistry continue to refine the chemical class, enhancing its potency and specificity. The study of ATF-7IP inhibitors represents a promising avenue in deciphering the complexities of gene regulation and chromatin dynamics, offering insights into the fundamental mechanisms governing cellular processes and providing a foundation for further exploration in the realm of molecular biology.
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Product Name | CAS # | Catalog # | QUANTITY | Price | Citations | RATING |
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Akt Inhibitor VIII, Isozyme-Selective, Akti-1/2 | 612847-09-3 | sc-202048 sc-202048A | 1 mg 5 mg | $204.00 $265.00 | 29 | |
Akti-1/2 is an Akt inhibitor, it can inhibit ATF-7IP by blocking the Akt signaling pathway. | ||||||
GSK 690693 | 937174-76-0 | sc-363280 sc-363280A | 10 mg 50 mg | $255.00 $1071.00 | 4 | |
GSK690693 is an Akt kinase inhibitor, it can inhibit ATF-7IP by blocking the Akt signaling pathway. |