Date published: 2025-12-24

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T-type Ca++ CP alpha 1G Inhibitors

Santa Cruz Biotechnology now offers a broad range of T-type Ca++ CP alpha 1G Inhibitors for use in various applications. This category of chemical inhibitors targets the T-type calcium channel alpha 1G subunit, a critical component in the regulation of calcium influx in neurons and other excitable cells. These inhibitors are essential for research focused on understanding calcium dynamics within the cellular environment, particularly how these dynamics influence neuronal excitability, rhythmicity, and overall cellular function. By selectively blocking the alpha 1G subunit, researchers can dissect its specific roles and contributions to calcium signaling pathways that are crucial for the functioning of nervous and cardiovascular systems. This targeted inhibition is particularly valuable in the study of neural networks and the mechanisms underlying neuronal oscillations and coupling. The ability to modulate these channels allows scientists to probe the fundamental aspects of neurophysiology, such as synaptic transmission and the generation of rhythmic firing patterns. Beyond neurological research, T-type Ca++ CP alpha 1G inhibitors are used in exploring the role of these channels in other physiological processes like muscle contraction and hormone secretion. This research aids in the broader understanding of how disturbances in calcium signaling can affect various biological systems, paving the way for advancements in non-clinical experimental methods and models. These inhibitors are not only pivotal in advancing basic scientific knowledge but also play a crucial role in the development of new techniques for studying cellular behavior. View detailed information on our available T-type Ca++ CP alpha 1G Inhibitors by clicking on the product name.
Product NameCAS #Catalog #QUANTITYPriceCitationsRATING

DHEA

53-43-0sc-202573
10 g
$109.00
3
(1)

DHEA, as a T-type Ca++ channel alpha 1G, exhibits unique gating properties that facilitate calcium influx in excitable tissues. Its interaction with lipid membranes alters channel conformation, enhancing permeability to calcium ions. This modulation influences cellular excitability and neurotransmitter release. The kinetics of DHEA's action are characterized by rapid activation and inactivation, contributing to transient calcium signaling that plays a crucial role in various physiological processes.