Sm N Activators

Small Nuclear Ribonucleoprotein Polypeptide N (SNRPN) is an essential gene found in the Prader-Willi syndrome (PWS) critical region in humans. It plays a key role in pre-mRNA splicing. SNRPN Activators would refer to a class of compounds that can modulate the activity of SNRPN, a gene involved in pre-mRNA splicing. The compounds listed above are selected based on their ability to influence the cellular processes and pathways that SNRPN is involved in. For instance, 5-Azacytidine, an inhibitor of DNA methylation, and Trichostatin A, a histone deacetylase inhibitor, can potentially affect the expression of SNRPN.

Other compounds, such as sodium butyrate, retinoic acid, and Dexamethasone can affect gene expression, cell differentiation, and immune responses, potentially influencing the function of SNRPN. Forskolin, genistein, and a range of DNA-interacting compounds (etoposide, doxorubicin, cisplatin, methotrexate, and daunorubicin) can affect various cellular processes and signaling pathways, potentially influencing the function or expression of SNRPN.