Date published: 2025-10-15

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BJ-TSA-9 Inhibitors

BJ-TSA-9 inhibitors are a class of chemical compounds that specifically target the BJ-TSA-9 protein or its associated pathways. BJ-TSA-9 is a small molecule often studied in the context of its ability to interact with cellular components involved in gene regulation and chromatin structure. While the precise role of BJ-TSA-9 may vary depending on the biological system under investigation, it is commonly linked to epigenetic modulation and transcriptional control, influencing how genes are expressed and how chromatin is remodeled within the cell. Inhibitors targeting BJ-TSA-9 are designed to block its functional activity, allowing researchers to better understand the molecular interactions and biological pathways associated with this compound. These inhibitors provide a valuable tool for examining the underlying mechanisms through which BJ-TSA-9 influences cellular processes.

BJ-TSA-9 inhibitors are small molecules engineered to bind to specific regions of the protein or disrupt its interaction with chromatin or transcriptional machinery. By doing so, these inhibitors prevent BJ-TSA-9 from exerting its effects on gene expression, chromatin dynamics, or other cellular functions. Researchers often use these inhibitors to study the role of BJ-TSA-9 in epigenetic regulation, transcriptional repression, or activation, as well as its involvement in broader cellular processes such as cell cycle regulation and differentiation. By inhibiting the activity of BJ-TSA-9, scientists can dissect its specific contributions to cellular signaling and gene regulatory networks, offering a deeper understanding of how this protein interacts with other molecular players in the cell. This makes BJ-TSA-9 inhibitors essential tools in experimental settings where precise control of transcriptional or epigenetic factors is required.

SEE ALSO...

Product NameCAS #Catalog #QUANTITYPriceCitationsRATING

Trichostatin A

58880-19-6sc-3511
sc-3511A
sc-3511B
sc-3511C
sc-3511D
1 mg
5 mg
10 mg
25 mg
50 mg
$149.00
$470.00
$620.00
$1199.00
$2090.00
33
(3)

Trichostatin A is a histone deacetylase inhibitor that could prevent BJ-TSA-9 transcription by maintaining a closed chromatin structure.

5-Azacytidine

320-67-2sc-221003
500 mg
$280.00
4
(1)

This DNA methyltransferase inhibitor could lead to the hypomethylation of the BJ-TSA-9 gene promoter, reducing its expression.

Actinomycin D

50-76-0sc-200906
sc-200906A
sc-200906B
sc-200906C
sc-200906D
5 mg
25 mg
100 mg
1 g
10 g
$73.00
$238.00
$717.00
$2522.00
$21420.00
53
(3)

Actinomycin D binds to DNA and could inhibit RNA polymerase from transcribing BJ-TSA-9 by intercalating DNA strands.

Cycloheximide

66-81-9sc-3508B
sc-3508
sc-3508A
100 mg
1 g
5 g
$40.00
$82.00
$256.00
127
(5)

Cycloheximide inhibits eukaryotic protein synthesis, potentially decreasing BJ-TSA-9 levels by preventing its translation.

α-Amanitin

23109-05-9sc-202440
sc-202440A
1 mg
5 mg
$260.00
$1029.00
26
(2)

Alpha-amanitin inhibits RNA polymerase II, which could reduce BJ-TSA-9 mRNA synthesis and, consequently, its protein levels.

Chloroquine

54-05-7sc-507304
250 mg
$68.00
2
(0)

Chloroquine can raise endosomal pH, which might hypothesize to interfere with the transcription factors needed for BJ-TSA-9 expression.

Camptothecin

7689-03-4sc-200871
sc-200871A
sc-200871B
50 mg
250 mg
100 mg
$57.00
$182.00
$92.00
21
(2)

Camptothecin is a topoisomerase I inhibitor that could hypothetically interrupt the unwinding of DNA required for BJ-TSA-9 transcription.

Mithramycin A

18378-89-7sc-200909
1 mg
$54.00
6
(1)

Mithramycin A binds to G-C rich DNA sequences, potentially blocking the binding sites and transcription of BJ-TSA-9.

DRB

53-85-0sc-200581
sc-200581A
sc-200581B
sc-200581C
10 mg
50 mg
100 mg
250 mg
$42.00
$185.00
$310.00
$650.00
6
(1)

DRB acts as an inhibitor of RNA polymerase II transcription elongation, which could suppress BJ-TSA-9 expression.

Flavopiridol Hydrochloride

131740-09-5sc-207687
10 mg
$311.00
(2)

Flavopiridol inhibits cyclin-dependent kinases, which could lead to a halt in the cell cycle and potentially downregulate BJ-TSA-9 expression.