SUCLA2 Antibody (F-2) is a mouse monoclonal IgG2b (kappa light chain) provided at 200 µg/ml
specific for an epitope mapping between amino acids 59-93 near the N-terminus of SUCLA2 of human origin
recommended for detection of SUCLA2 of mouse, rat and human origin by WB, IP, IF and ELISA; also reactive with additional species, including bovine
m-IgG2b BP-HRP and m-IgGκ BP-HRP are the preferred secondary detection reagents for SUCLA2 Antibody (F-2) for WB applications. These reagents are now offered in bundles with SUCLA2 Antibody (F-2) (see ordering information below).
Every item is shipped based on the best shipping method assessed for the temperature requirements of that specific item. Items are grouped and shipped together whenever
possible, and a separate shipping charge will be included for each shipping method required. Shipping charges listed below are from our US warehouses to the Contiguous US,
Alaska, Hawaii, Canada and Puerto Rico. Shipping charges for countries outside the US and Canada will be determined once order has been received
Please note: We can not ship to PO boxes
Express Blue Ice
Express Dry Ice
Animal Health Prescription Item
SHIPPING METHODS & CHARGES
Ships via FedEx Ground to Contiguous US, Alaska, Canada, Monday through Friday. This method is used for less temperature sensitive items such as lab ware and animal
health products, bulky and/or heavy items
Labware ships FedEx Ground free of charge to the contiguous US
SUCLA2 (succinate-CoA ligase, ADP-forming, beta subunit), also known as A-BETA, SCS-betaA or renal carcinoma antigen NY-REN-39, is a 463 amino acid mitochondrial matrix enzyme that belongs to the succinate/malate CoA ligase beta subunit family. Widely expressed, SUCLA2 dimerizes with the SCS alpha subunit to form SCS-A, an essential component of the tricarboxylic acid cycle. Defects in SUCLA2 may be involved in a group of autosomal recessive disorders known as Mitochondrial DNA depletion syndromes (MDSs) that are characterized by a decrease in mitochondrial DNA copy numbers in affected tissues. Progressive external ophthalmoplegia (PEO), ataxia-neuropathy and mitochondrial neurogastrointestinal encephalomyopathy (MNGIE) may also be associated with mutations in SUCLA2. Two isoforms of SUCLA2 exist due to alternative splicing events.
For Research Use Only. Not Intended for Diagnostic or Therapeutic Use.