Citreoviridin, a mycotoxin, was initially isolated from Penicillium citreoviride. This agent displays the ability to potently inhibit mitochondrial ATPases via uncompetitive inhibition of ATP hydrolysis. Additional research shows that Citreoviridin can inhibit ATP-driven reduction of NAD+ by succinate and ATP driven NAD transhydrogenase in ox hearts.
1. Linnett, P.E., et al. 1978. Biochem. J. 170: 503-510. PMID: 148274 2. Satre, M., et al. 1980. J. Bacteriol. 142: 768-776. PMID: 6445896 3. Gause, E.M., et al. 1981. J. Biol. Chem. 256: 557-559. PMID: 6450205 4. Sayood, S.F., et al. 1989. Arch. Biochem. Biophys. 270: 714-721. PMID: 2523213
Soluble in ethanol, chloroform, DMSO, and dichloromethane. Insoluble in water.
Store at -20° C
585.12° C at 760 mmHg (Predicted)
1.19 g/cm3 (Predicted)
n20D 1.57 (Predicted)
For Research Use Only. Not Intended for Diagnostic or Therapeutic Use.
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Rated 5 out of
Sayood et alSayood et al. (PubMed ID 2523213) showed that Citreoviridin was an uncompetitive inhibitor of ATP hydrolysis and a noncompetitive inhibitor of ATP synthesis catalyzed by membrane-bound F1-ATPase in been heart mitochondria. -SCBT Publication Review
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