Date published: 2025-12-24

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T-type Ca++ CP alpha 1H Inhibitors

Santa Cruz Biotechnology now offers a broad range of T-type Ca++ CP alpha 1H Inhibitors for use in various applications. T-type Ca++ CP alpha 1H inhibitors target a specific subclass of calcium channels that are critical in the regulation of electrical activity in the heart and neurons. These inhibitors are valuable tools in basic research and physiology studies, helping scientists to understand the role of these channels in controlling heart rhythms and neuronal excitability. The ability to selectively inhibit T-type calcium channels enables researchers to dissect the contributions of these channels to various physiological processes, such as cardiac function, neuronal firing, and rhythmicity in pacemaker cells. By modulating the activity of these channels, researchers can explore the underlying mechanisms of electrical signaling and its impact on cellular behavior in a non-clinical setting. This research has profound implications for understanding fundamental biological processes and can lead to the development of new experimental models for studying cellular excitability and signaling. Furthermore, these inhibitors serve as a bridge to explore how disturbances in calcium signaling can affect tissue function, providing insights into the basic science of cellular communication and electrophysiology. In the broader scientific community, T-type Ca++ CP alpha 1H inhibitors have facilitated advancements in the study of electrophysiology by enabling precise manipulation of calcium-dependent processes in various cell types, contributing significantly to our understanding of cellular dynamics. View detailed information on our available T-type Ca++ CP alpha 1H Inhibitors by clicking on the product name.
Product NameCAS #Catalog #QUANTITYPriceCitationsRATING

DHEA

53-43-0sc-202573
10 g
$109.00
3
(1)

DHEA, functioning as a T-type Ca++ channel alpha 1H, demonstrates distinctive voltage-dependent activation, allowing for selective calcium entry in response to membrane depolarization. Its binding affinity to specific sites on the channel influences the kinetics of calcium ion flow, resulting in a unique pattern of intracellular signaling. This channel's behavior is marked by a slower inactivation phase, which can prolong calcium currents, thereby impacting cellular responses and excitability in a nuanced manner.

L-Ascorbic acid, cell culture

50-81-7sc-394304
100 mg
$94.00
1
(2)

L-Ascorbic acid, as a T-type Ca++ channel alpha 1H, exhibits unique modulation of calcium ion dynamics through its interaction with the channel's gating mechanisms. This compound enhances calcium influx during depolarization, influencing the channel's activation threshold. Its role in stabilizing the channel conformation contributes to altered calcium signaling pathways, promoting distinct intracellular responses. The kinetics of calcium entry are characterized by a gradual onset, affecting cellular excitability and signaling cascades.